Knowledge gained through this course will enhance learners’ understanding of cancer biology, the mechanism of action of systemic cancer treatments, and the use of biomarkers to select the best treatments for each patient and as a result empower staff to feel confident when communicating with patients and their families, and with clinical and non-clinical colleagues.
This course describes how bowel cancer comes about, and explains the reasons why some patients might be given treatments such as EGFR-targeted antibodies (cetuximab and panitumumab), angiogenesis inhibitors (bevacizumab and regorafenib), or immune checkpoint inhibitors (pembrolizumab) as part of their treatment.
This course provides an overview of the targeted therapies and immunotherapies given to some people with oesophageal, stomach, pancreatic or liver cancer, or cancers of the biliary system
This course explains why breast cancer arises, the different ways that the DNA in our cells gets damaged and the targeted treatments that match a few of the known mutations. It also explores the science behind hormone therapies, targeted therapies, and immunotherapies given to patients with hormone receptor-positive, HER2-positive, and triple-negative breast cancer.
Haematological cancers differ from solid tumours in many ways: in the mutations that drive them, their interactions with their environment, and the treatments that are effective against them. This course introduces the biology, behaviours and genetics of this diverse group of diseases with a focus on the features we can now target and the mechanisms of action of the various treatments
During this introduction to the science behind a wide variety of systemic treatments given to people with prostate, bladder, or kidney cancer, Dr Elaine Vickers will discuss current hormone therapies, immunotherapy and targeted treatments.
A focus on modern systemic treatments for non-small cell lung cancer (NSCLC) as well as the gene faults that drive many NSCLCs; the targeted treatments that exploit these faults, such as inhibitors of EGFR, ALK, ROS1, B-Raf, HER2, MET, and RET; the immune system and checkpoint inhibitor group of immunotherapies, including PD-1, PD-L1 and CTLA-4 targeted antibody therapies